Latest Research
2025
Liparulo, I., Bazhin, A., Van Wyhe, G. K., Gunawan, A. L., Dadina, N., Kwon, J. H., Schepartz, A., Goun, E., & Stahl, A. (2025). A clickable CoQ imaging probe reveals that cellular uptake and lysosomal trafficking depend on CD36 and NPC1. Redox Biology, 103936. https://doi.org/10.1016/j.redox.2025.103936
He, Y., Ling, L., Dempsey, G., Kalindjian, N., Verghese, Z. A., Chen, E., Liparulo, I., Cho, I., Tran, V., Kumar, S., & Stahl, A. (n.d.). Bioenergetic responses to β-adrenergic stimulation in beige adipocytes depend on actomyosin-driven forces. bioRxiv.
Gunawan AL, Liparulo I, Stahl A. Mammalian mitohormesis: from mitochondrial stressors to organismal benefits. EMBO J. 2025 Oct;44(20):5640-5661. doi: 10.1038/s44318-025-00549-3. Epub 2025 Sep 8. PMID: 40926120; PMCID: PMC12528382.
He, Y., Ling, L., Dempsey, G., Kalindjian, N., Verghese, Z. A., Chen, E., Liparulo, I., Cho, I., Tran, V., Kumar, S., & Stahl, A. (n.d.). Bioenergetic responses to β-adrenergic stimulation in beige adipocytes depend on actomyosin-driven forces. bioRxiv.
2024
Qi, L., Groeger, M., Sharma, A. et al. Adipocyte inflammation is the primary driver of hepatic insulin resistance in a human iPSC-based microphysiological system. Nat Commun 15, 7991 (2024). https://doi.org/10.1038/s41467-024-52258-w
Shihadih, D., Wang, X., Zushin, P.-J. H., Khodakivskyi, P., Park, H. M., Tso, E., Shiblak, J., Misic, A., Louie, S. M., Ward, C., Hellerstein, M., Nomura, D. K., Goun, E., Urigo, F., Calvisi, D. F., Chen, X., & Stahl, A. (2024). FATP5 is indispensable for the growth of intrahepatic cholangiocarcinoma. Metabolism, 155, 155927. https://doi.org/10.1016/j.metabol.2024.155927
Chang, CF., Gunawan, A.L., Liparulo, I. et al. Brown adipose tissue CoQ deficiency activates the integrated stress response and FGF21-dependent mitohormesis. EMBO J 43, 168–195 (2024). https://doi.org/10.1038/s44318-023-00008-x
Qi, L., Matsuo, K., Pereira, A., Lee, Y. T., Zhong, F., He, Y., Zushin, P.-J. H., Gröger, M., Sharma, A., Willenbring, H., Hsiao, E. C., & Stahl, A. (n.d.). Human iPSC-derived proinflammatory macrophages cause insulin resistance in an isogenic white adipose tissue microphysiological system. Small.